Blood sugar
76 supplements analyzed and ranked by clinical evidence.
Supplement Evidence
HbA1c ↓, 0.5%
Postmeal glucose ↓
Insulin resistance ↓
Alanine↓ glucose↓
Alanine↑ glucose↑
Ketosis shifts alanine
K↓, glucose↓
insulin sensitivity↑, glucose↓
↓ HOMA-IR, glucose
↓ HbA1c, FPG
↓ insulin, +sensitivity
Lower glucose, better sensitivity
Higher glucose, worse sensitivity
Blood sugar ↓
Insulin sensitivity ↑
Lower glucose/HbA1c
Lower insulin resistance
Lower glucose/HbA1c
Better insulin resistance
Lower post-meal spikes
Post-meal glucose ↓
FPG/HbA1c ↓
Lower fasting glucose
HbA1c -0.83%
Lower glucose/insulin
Restored flexibility
+24% carnitine
Lower glucose, HOMA‑IR
+ glucose disposal
Butyrate↑ glucose↓
AUC−36 mg/dL
Lower glucose, HOMA-IR
−0.5 mmol/L FPG
Post-meal glucose, insulin
Lower glucose/HbA1c
Improve insulin resistance
Glucose↓ insulin↓
HbA1c↓ FPG↓
Post-meal glucose↓ insulin↓
PPG ↓ 18-39%
Insulin sens ↑; HbA1c ↓
FBG/2h glucose ↓
↑ insulin, ↓ glucose
↓ insulin resistance
Lower fasting glucose −27%
Better post‑meal glucose, insulin
↓ fasting/2h glucose
↔/↑ thiamine status
A1c↓ glucose↓
Akkermansia↑ endotoxin↓
Postmeal glucose ↓14-31%
Fasting glucose/HbA1c ↓
Postmeal insulin ↓12-36%
Fasting glucose ↓ ~9-12
HbA1c ↓ ~0.3-0.8%
Glucose ↓; insulin ↓1.43
Postmeal glucose ↓; insulin ↑
Insulin ↓, HOMA-IR ↓
Glucose ↓11.8 mg/dL
HbA1c ↓0.9%
Post-meal glucose ↓
HbA1c/FBS ↓
Lower post-meal glucose
↓ glucose/insulin peaks
More fermentation, lower HGP
Blood sugar ↓
HbA1c/FPG ↓
Lower fasting glucose
HbA1c -1.15%
Early insulin +100%
Glucose iAUC -61%
Diabetes incidence -41%
↓ glucose AUC
↑ insulin/GLP-1
−14% glucose AUC
Lower blood sugar
~ modest decrease
↓ Blood sugar
↓ Blood sugar -3.1%
↓ Blood sugar
Lower blood sugar
HbA1c -0.75%
Improved insulin sensitivity
Glucose/insulin ↓
+27% ISI
HbA1c −0.5%
↓ FPG, HbA1c
↓ PPG, GI
↓ FPG/HbA1c
↓ HOMA-IR / MG
Lower FPG/HbA1c (~20%, ~12%)
Lower FBS, insulin dose
Improved insulin resistance
Glucose↓, HOMA↓
HbA1c−1.1%
Glucose control −24 mg/dL
HbA1c −0.8%
Postprandial glucose −14%
↓ glucose spikes
↓ insulin response
Glucose ↓57%
Glucose ↓37%
Postmeal glucose ↓
Insulin timing ↑
HOMA-IR ↓ 21%
Blood sugar: GI −34%
Blood sugar: post‑meal glucose −16%
↓ insulin resistance
↓ fasting glucose −0.2 mmol/L
↑ GLP-1, SCFAs
↓ post-meal glucose
↓ fasting/HbA1c glucose
↓ insulin spikes
Blood sugar ↓
Blood sugar ↓ +1.25
Blood sugar ↔ HbA1c
Lower fasting glucose
Lower post-meal insulin
Lower fasting glucose, HbA1c
Lower insulin resistance ~30%
Higher GLP-1, lower glucose
HbA1c, glucose ↓
Postmeal glucose/insulin ↓
Gut diversity ↑
Glucose ↓; insulin ↓
-8% post-meal glucose
Insulin sensitivity +15%
Glucose, insulin AUC ↓
Fasting/post-meal sugar, HbA1c ↓
FBG −27 mg/dL, HbA1c −1.49%
Fasting glucose ↓; HbA1c −0.7%
Metabolic markers ↓; LDL ↓
Lower fasting glucose
Improved insulin resistance
Lower fasting glucose, HOMA (-11.7%)
Lower HbA1c, insulin resistance
Less sugar damage
↓AGEs; −2.45 CRP
Lower post-meal glucose
~10 mg/dL AUC↓
No glucose rise
-lower postmeal glucose
↓ HOMA-IR, ALT
↓ HOMA-IR, insulin
Fasting glucose -27%
Postprandial glucose -20%
Early insulin -33%
Glucose -1.3 mmol/L
HbA1c -0.4%
Insulin sensitivity +33%
Lower fasting glucose
Lower post-load glucose
Lower post-meal glucose
HbA1c -0.27%
BMI, visceral fat ↓
Subcutaneous fat no change
Lower fasting/postprandial glucose
−LDL, −insulin AUC
↓ insulin resistance (HOMA-IR)
↑ adiponectin (+20%)
Asprosin↓, MCP-1↓
Adiponectin↑, stronger with training
Glucose ↓ HbA1c −2.0
HOMA-IR ↓; BMI ↓
FBS -10.57 mg/dL
Lower blood sugar
Post-meal glucose↓
Insulin AUC↓
Lower glucose; better insulin sensitivity
↓ glucose AUC
Lower post-meal glucose
40g dose effect
Butyrate +17.4%
Insulin sensitivity ↑
Triglycerides ↓
Triglycerides/VLDL ↓
Carboxylase markers ↑
↓ insulin, −36.5%
↓ HOMA-IR, −0.8